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It is extremely important to begin taking the anti-androgen (Eulexin, Casodex, Nilandron) ten days to two weeks before the first shot of the LHRH agonist (Zoladex, Lupron, Viadur).
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LHRH agonists are associated with acute-on-chronic effect in 4% to 10% of patients treated with standard LHRH therapy.
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These patients are then often given salvage radiation therapy to the prostate tumor bed.
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They bring about an initial release in testosterone, followed by a fall in the levels, with final effects similar to castration.
However, this effect is temporary and after a median of 18-24 months, there is disease progression, heralded by rising serum prostate-specific antigen (PSA), increasing tumor size, new metastatic spread and disease-related symptoms.
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Effective treatment of this avascular necrosis has not been determined, although conservative management with penicillin-type antibiotics and chlorhexidine rinses seems effective.
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Degarelix also provided rapid suppression of testosterone levels (Figure 1).
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As more patients with advanced prostate cancers receive antiandrogen chemotherapy, more cases of drug-induced lung diseases may be encountered.
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AR(cs)) (harbors a mixture of exogenous wild-type AR and endogenous mutant AR (T877A)) and cells propagated in Iscove's or RPMI media supplemented with 10% fetal bovine serum, or during the assay with 10% charcoal-stripped, dextran-treated fetal bovine serum (CSS).
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Given this discordance, as of August 2010, all testing was conducted centrally at MSKCC for standardization of methods.
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If you have not yet chosen a treatment (or a physician), you can have your urologist determine the volume of your prostate and monitor the progress so that you can avoid reducing the size beyond that which is ideal for the procedure.
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You may receive other cancer medicines at the same time.
Other secondary endpoints were the changes in serum levels of PSA, testosterone, estradiol, LH, FSH, and SHBG at 3 months relative to baseline (trial entry).
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In addition to shrinking tumor, increasing organ confinement, and decreasing the incidence of positive margins, neoadjuvant therapy has also been theorized to treat occult regional and systemic micrometastasis, with the ultimate goal being improved long-term, disease-free survival.
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Although more dissections that were difficult were reported with NAD therapy in this study, no operative complications occurred in the NAD-treated group, whereas 6 intraoperative injuries were reported in patients who underwent surgery alone.
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We have shown that KUCaP derived from clinical materials with W741C mutant AR maintained the mutation in the xenografts and that the response of KUCaP to bicalutamide in vivo was in concordance with the results of transactivation assays using W741C mutant AR.
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